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Emergent Hebbian Dynamics in Regularized Learning: A Theoretical Analysis

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[Submitted on 23 May 2025 (v1), last revised 28 May 2026 (this version, v3)]

8d ago· 2 min readenInsight

Summary

This research paper investigates whether observed Hebbian/anti-Hebbian plasticity in synaptic updates necessarily implies an underlying Hebbian computation mechanism. The authors identify an alternative, emergent route: near stationarity, L2 weight decay can drive the learning-signal component of many update rules to align with a Hebbian direction, with alignment increasing with decay strength. This Hebbian-like signature can arise even for non-learning or random update rules, and stochastic noise can induce anti-Hebbian alignment. The findings suggest that emergent Hebbian signatures can coexist with genuine Hebbian plasticity, complicating the interpretation of synaptic measurements and motivating new experiments to distinguish mechanistic from emergent Hebbian computation.

Key quotes

· 4 pulled
We identify an alternative, emergent route: near stationarity, L2 weight decay generically drives the learning-signal component of many update rules to align with a Hebbian direction, with alignment increasing monotonically with decay strength.
This Hebbian-like signature is not specific to SGD and can arise even for non-learning or random update rules long before learning has ceased.
Stochastic noise in the learning signal can induce anti-Hebbian alignment, yielding a simple tradeoff with weight decay and a phase boundary in regression settings.
These mechanisms do not replace standard Hebbian theory; they can coexist with genuine Hebbian plasticity and complicate the interpretation of synaptic measurements.
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Hebbian and anti-Hebbian plasticity are widely observed in the brain and are classically modeled as mechanistic, local homosynaptic rules stabilized by homeostatic constraints. This raises an identifiability question: does observing Hebbian/anti-Hebbian s

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